Substance reference

Vitamin E

Vitamins EFSANIH ODS / IOM-DRI (NASEM) 13 products in Stacky

What the European Food Safety Authority and the United States National Institutes of Health have published about vitamin E intake, and what the catalogue records. Reference figures and citations only.

Stacky is an organisational tool, not a medical device. It does not provide medical advice, and nothing on this page says what vitamin E does. Talk to a doctor or pharmacist before starting, stopping or changing what you take.

Published intake references

Each figure is one authority’s published value, with the kind of figure and the people it applies to as that authority stated them. Where the EU and the US differ, both are shown and neither is averaged or converted. Only a tolerable upper intake level sets a ceiling. A recommended or adequate intake is context, not a limit.

European Union — EFSA

Adequate intake 11 mg adult females 18+
Applies to one chemical form only: as alpha-tocopherol; EFSA publishes in mg alpha-tocopherol, not IU. 1 IU = 0.67 mg natural (RRR-) or 0.45 mg synthetic (all-rac-) alpha-tocopherol.
EFSA →
Adequate intake 13 mg adult males 18+
Applies to one chemical form only: as alpha-tocopherol; EFSA publishes in mg alpha-tocopherol, not IU. 1 IU = 0.67 mg natural (RRR-) or 0.45 mg synthetic (all-rac-) alpha-tocopherol.
EFSA →
Tolerable upper intake level 300 mg adults 18+, TOTAL intake from all dietary sources. Originally set by the SCF in 2003 (NOAEL 800 IU/day = 540 mg/day, uncertainty factor 2…Our record of this statement is cut short; the authority’s document has it in full.
Applies to one chemical form only: as alpha-tocopherol - the only physiologically active vitamer; EFSA restricts the UL to alpha-tocopherol. EFSA publishes vitamin E in mg alpha-tocopherol, NOT in IU. Label conversion: 1 IU = 0.67 mg RRR-alpha-tocopherol (natural, d-) = 0.45 mg all-rac-alp
EFSA →

United States — NIH ODS / IOM

Recommended daily intake (RDA / PRI) 15 mg adults 19+, both sexes
Applies to one chemical form only: as alpha-tocopherol; published in mg, not IU. 1 IU = 0.67 mg natural (RRR-) or 0.45 mg synthetic (all-rac-) alpha-tocopherol, so 15 mg = approx. 22 IU natural.
NIH ODS / IOM-DRI (NASEM) →
Tolerable upper intake level 1000 mg adults 19+; applies to SYNTHETIC/SUPPLEMENTAL forms obtained from supplements, fortified foods, or a combination of the two - not to alpha-tocopherol naturally present in food
Applies to one chemical form only: as alpha-tocopherol; applies to any form of supplemental alpha-tocopherol. The IOM/NASEM publishes in mg alpha-tocopherol, NOT in IU. 1 IU = 0.67 mg RRR-alpha-tocopherol (natural, d-) = 0.45 mg all-rac-alpha-tocopherol (synthetic, dl-), so the 1,000 mg UL
NIH ODS / IOM-DRI (NASEM) →

How it is recorded

What the app records so it can schedule a dose. None of it is advice.

Forms recorded: Softgel, Capsule.

Food: the app records vitamin E as taken with food.

Recorded interactions

What a named source says about vitamin E taken alongside another substance: absorption and nutrient status only. Each row links to its source. A row without one is not published, and neither is one whose source description names a disease or clinical condition: 1 such row for Vitamin E stays in the app. None of these says whether a combination is good or bad.

RelationshipWhat the source saysSource
Documented relationship with Omega-3 The amount of vitamin E needed to prevent lipid peroxidation rises with the amount of polyunsaturated fat consumed, which is why many fish oil products include vitamin E. LPI notes that high vitamin E intakes have not been shown to lower markers of oxidative damage when EPA and DHA intakes increase, though some experts still recommend raising vitamin E alongside omega-3.
Scope the source gave: increases with the amount of polyunsaturated fat consumed.
Linus Pauling Institute
Checked 6 Aug 2026
Documented relationship with Selenium Selenium acts through selenoenzymes alongside vitamin E, regenerating it from its oxidised form. The two are frequently formulated together for this reason. Linus Pauling Institute
Checked 6 Aug 2026
Documented relationship with Vitamin C Vitamin C regenerates other antioxidants in the body, including alpha-tocopherol. In a trial in smokers and non-smokers, vitamin C supplementation reduced the rate of vitamin E loss from plasma. NIH ODS
Checked 6 Aug 2026
Reduces absorption or status of Vitamin K2 Large doses of vitamin E antagonise vitamin K, apparently by inhibiting vitamin K-dependent carboxylase activity. In adults with normal coagulation status, 1,000 IU a day of vitamin E for 12 weeks decreased carboxylation of prothrombin, a vitamin K-dependent protein.
Scope the source gave: large doses of vitamin E; one study used 1,000 IU a day for 12 weeks.
Linus Pauling Institute
Checked 6 Aug 2026

Indexed studies

Stacky has 5 papers indexed against vitamin E, and none of them is published here. A study reaches this page only if its own title names Vitamin E and does not name a disease or clinical condition. None of the 5 passes that rule. They remain in the app. How this list is built →

About this page

Stacky is a supplement tracking app. It renders what you take and what published sources say about it; it does not tell you what to take. Every figure on this page names the authority that published it and links to that authority’s own page.

Not on this page: a dose of our own, a grade, a verdict on whether vitamin E works, or any statement that it treats, prevents or improves a condition. How these pages are built →

Figures are read from the authorities’ published tables and can go out of date when those tables are revised. If one here is wrong, tell us at support@stacky-app.com and we will check it against the source.